About MEAJO | Editorial board | Search | Ahead of print | Current Issue | Archives | Instructions to authors | Online submission | Subscribe | Advertise | Contact | Login 
Middle East African Journal of Ophthalmology Middle East African Journal of Ophthalmology
Users Online: 541   Home Print this page Email this page Small font sizeDefault font sizeIncrease font size
Year : 2010  |  Volume : 17  |  Issue : 3  |  Page : 270-274

Idiopathic choroidal neovascularisation as the inaugural sign of multiple evanescent white dot syndrome

1 Centre for Specialized Ophthalmic Care (COS), University of Genova, Genova; Eye Clinic, Department of Neurosciences, Ophthalmology and Genetics, University of Genova, Genova, Italy
2 Centre for Specialized Ophthalmic Care (COS), University of Genova, Genova, Italy; University of Lausanne, Lausanne, Switzerland

Correspondence Address:
Carl Peter Herbort
Centre for Specialized Ophthalmic Care, Centre d'Ophtlamologie Spécialisée (COS) 6, rue de la Grotte, 1003 Lausanne, Switzerland

Login to access the Email id

Source of Support: None, Conflict of Interest: None

DOI: 10.4103/0974-9233.65490

Rights and Permissions

We report a case of multiple evanescent white dot syndrome (MEWDS) that presented with putative idiopathic choroidal neovascularisation (ICNV) before showing angiographic signs typical of MEWDS. A 16-year-old male presented with unilateral metamorphopsias and visual loss in his left eye. ICNV with subretinal hemorrhage was diagnosed and treated with intravitreal Avastin® . Fifteen days later, regression of choriodal neovascularization (CNV) was documented together with the appearance of fluorescein angiography (FA) and indocyanine green angiography (ICGA) signs typical for MEWDS, that included faint mottled FA hyperfluorescence in the mid-peripheral fundus, irregularly shaped mid-peripheral ICGA dark areas in the intermediate angiographic phase that were clearly delineated in the late phase as well as peripapillary hypofluorescence. Fundus examination appeared completely normal during the follow-up except for the CNV hemorrhage noted at the initial visit. This case demonstrates the need to consider ICNV as a diagnosis of exclusion until inflammatory causes have been eliminated. In this case, the underlying occult inflammatory condition would have been missed without the ICGA data that clearly showed signs of MEWDS that was supported by FA findings.

Print this article     Email this article
 Next article
 Previous article
 Table of Contents

 Similar in PUBMED
   Search Pubmed for
   Search in Google Scholar for
 Citation Manager
 Access Statistics
 Reader Comments
 Email Alert *
 Add to My List *
 * Requires registration (Free)

 Article Access Statistics
    PDF Downloaded152    
    Comments [Add]    
    Cited by others 4    

Recommend this journal